Karoderin

Karoderin Uses, Dosage, Side Effects, Food Interaction and all others data.

An antimuscarinic agent that inhibits gastric secretion at lower doses than are required to affect gastrointestinal motility, salivary, central nervous system, cardiovascular, ocular, and urinary function. It promotes the healing of duodenal ulcers and due to its cytoprotective action is beneficial in the prevention of duodenal ulcer recurrence. It also potentiates the effect of other antiulcer agents such as cimetidine and ranitidine. It is generally well tolerated by patients.

Karoderin belongs to a group of medications called antispasmodics/anticholinergics. These medications are used to relieve cramps or spasms of the stomach, intestines, and bladder. Karoderin is used to treat duodenal or stomach ulcers or intestine problems. It can be used together with antacids or other medicine in the treatment of peptic ulcer. It may also be used to prevent nausea, vomiting, and motion sickness.

Table Of contents

• Karoderin
• Uses
• Dosage
• Side Effect
• Precautions
• Interactions
• Uses during Pregnancy
• Uses during Breastfeeding
• Accute Overdose
• Food Interaction
• Half Life
• Volume of Distribution
• Clearance
• Interaction With other Medicine
• Contradiction
• Storage

Trade Name	Karoderin
Generic	Pirenzepine
Pirenzepine Other Names	Pirenzepin, Pirenzepina, Pirenzépine, Pirenzepine, Pirenzepinum
Type
Formula	C19H21N5O2
Weight	Average: 351.4023 Monoisotopic: 351.169524941
Groups	Approved
Therapeutic Class
Manufacturer
Available Country
Last Updated:	September 19, 2023 at 7:00 am

Uses

Karoderin is an antimuscarinic agent used to treat peptic ulcers, gastric ulcers, and duodenal ulcers.

For the treatment of peptic ulcer, gastric ulcer, and duodenal ulcer.

Karoderin is also used to associated treatment for these conditions: Gastric Ulcer, Small intestine ulcer

How Karoderin works

Karoderin is a muscarinic receptor antagonist and binds to the muscarinic acetylcholine receptor. The muscarinic acetylcholine receptor mediates various cellular responses, including inhibition of adenylate cyclase, breakdown of phosphoinositides and modulation of potassium channels through the action of G proteins.

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