Pirenzepine Hydrochloride Nichi-Iko
Pirenzepine Hydrochloride Nichi-Iko Uses, Dosage, Side Effects, Food Interaction and all others data.
An antimuscarinic agent that inhibits gastric secretion at lower doses than are required to affect gastrointestinal motility, salivary, central nervous system, cardiovascular, ocular, and urinary function. It promotes the healing of duodenal ulcers and due to its cytoprotective action is beneficial in the prevention of duodenal ulcer recurrence. It also potentiates the effect of other antiulcer agents such as cimetidine and ranitidine. It is generally well tolerated by patients.
Pirenzepine Hydrochloride Nichi-Iko belongs to a group of medications called antispasmodics/anticholinergics. These medications are used to relieve cramps or spasms of the stomach, intestines, and bladder. Pirenzepine Hydrochloride Nichi-Iko is used to treat duodenal or stomach ulcers or intestine problems. It can be used together with antacids or other medicine in the treatment of peptic ulcer. It may also be used to prevent nausea, vomiting, and motion sickness.
Trade Name | Pirenzepine Hydrochloride Nichi-Iko |
Generic | Pirenzepine |
Pirenzepine Other Names | Pirenzepin, Pirenzepina, Pirenzépine, Pirenzepine, Pirenzepinum |
Type | |
Formula | C19H21N5O2 |
Weight | Average: 351.4023 Monoisotopic: 351.169524941 |
Groups | Approved |
Therapeutic Class | |
Manufacturer | |
Available Country | Japan |
Last Updated: | September 19, 2023 at 7:00 am |
Uses
Pirenzepine Hydrochloride Nichi-Iko is an antimuscarinic agent used to treat peptic ulcers, gastric ulcers, and duodenal ulcers.
For the treatment of peptic ulcer, gastric ulcer, and duodenal ulcer.
Pirenzepine Hydrochloride Nichi-Iko is also used to associated treatment for these conditions: Gastric Ulcer, Small intestine ulcer
How Pirenzepine Hydrochloride Nichi-Iko works
Pirenzepine Hydrochloride Nichi-Iko is a muscarinic receptor antagonist and binds to the muscarinic acetylcholine receptor. The muscarinic acetylcholine receptor mediates various cellular responses, including inhibition of adenylate cyclase, breakdown of phosphoinositides and modulation of potassium channels through the action of G proteins.
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