Winstrol Depot

Winstrol Depot Uses, Dosage, Side Effects, Food Interaction and all others data.

Winstrol Depot is a synthetic anabolic steroid with therapeutic uses in treating hereditary angioedema. Winstrol Depot is derived from testosterone, and has been abused by several high profile professional athletes.

Winstrol Depot is a synthetic anabolic-androgenic steroid (AAS), which promotes cell growth (anabolism) and development/maintenance of masculine characteristics (androgenism).

Winstrol Depot
Uses
Dosage
Side Effect
Precautions
Interactions
Uses during Pregnancy
Uses during Breastfeeding
Accute Overdose
Food Interaction
Half Life
Volume of Distribution
Clearance
Interaction With other Medicine
Contradiction
Storage

Trade Name	Winstrol Depot
Availability	Discontinued
Generic	Stanozolol
Stanozolol Other Names	Androstanazol, Androstanazole, Estanozolol, Stanozolol
Related Drugs	Orladeyo, danazol, Firazyr, Haegarda, Ruconest, Berinert, Danocrine, Cinryze, Kalbitor
Type
Formula	C₂₁H₃₂N₂O
Weight	Average: 328.4916 Monoisotopic: 328.251463656
Groups	Approved, Vet approved
Therapeutic Class
Manufacturer
Available Country
Last Updated:	September 19, 2023 at 7:00 am

Uses

Winstrol Depot is an anabolic steroid used to manage hereditary angioedema.

Winstrol Depot is a synthetic anabolic steroid with therapeutic uses in treating C1-inhibitor deficient hereditary angioedema. C1-inhibitor is a protease that inhibits the complement system (part of the innate immune system), a biochemical chain of reactions which assists the body in removing pathogens from the body. Winstrol Depot may help control attacks of hereditary angioedema. Winstrol Depot can be administered orally or intramuscularly.

Winstrol Depot is also used to associated treatment for these conditions: Hereditary Angioedema

How Winstrol Depot works

Winstrol Depot binds to androgen receptors, such as membrane bound receptor proteins LAGS and stanozolol-binding protein (STBP).

Winstrol Depot Cholesterol interaction

[Major] Androgenic anabolic steroids may adversely affect serum lipids, including lowering HDL and elevating LDL levels.

These changes can be marked, particularly with the 17-alpha-alkyl derivatives (i.e., fluoxymesterone, methyltestosterone, oxandrolone, oxymetholone, and stanozolol), and may significantly impact the risk of atherosclerosis and coronary artery disease.

Patients with preexisting hyperlipoproteinemia may require closer monitoring during therapy with androgenic agents, and adjustments made accordingly in their lipid-lowering regimen.

Androgen therapy should be administered cautiously in patients with coronary artery disease or a history of ischemic heart disease.